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Notes on Indian
Venomous Snakes and First Aid
Ashok Captain
Of the 275 or more species
of snakes found in India, we still have no real idea how many
are capable of causing human fatalities.
The commonly used description of the “Big 4” is outdated. Now we
refer to “Snakes of Medical Importance”.
It is beyond the scope of this note to provide an identification
key for the venomous snakes.
Species found within the Western Ghats as well as in regions of
Maharashtra not in the Western Ghats, that are known to have
caused human fatalities are:
| No |
Common name |
Scientific name |
Currently known
distribution |
| 1 |
Common Krait |
Bungarus caeruleus
|
Most of mainland India.
Probably absent from Assam upwards. |
| 2 |
Banded Krait |
Bungarus fasciatus
|
West Bengal, Bihar, Orissa,
Assam onwards to Arunachal Pradesh. Maharashtra (Gadchiroli
district), parts of Madhya Pradesh and Uttar Pradesh.
Possibly Uttaranchal. |
| 3 |
Wall’s Sind Krait |
Bungarus sindanus walli
|
Uttar Pradesh (Faizabad),
Maharashtra (Pune, Sholapur, Amravati), Bihar (Gaya,
Purnia), Bengal (Midnapore) |
| 4 |
Russell’s Viper |
Daboia russelii
|
Throughout India. |
| 5 |
Saw-scaled Viper |
Echis carinatus
|
Throughout India. |
| 6 |
Hump-nosed Pit Viper |
Hypnale hypnale
|
Western Ghats (northernmost
known limit – Belgaum). |
| 7 |
Common Cobra |
Naja naja
|
Throughout mainland India
except N. E. |
| 8 |
King Cobra |
Ophiophagus hannah
|
Western Ghats (Goa, Karnataka,
Kerala, Tamil Nadu. Uttar Pradesh (Terai), Uttaranchal,
Bihar, Orissa, West Bengal onwards to Arunachal Pradesh.
Andaman Islands. |
Sea snakes have not been dealt with.
All “true” Sea snakes (Family Hydrophidae) have flat, oar-like
tails and though capable of causing human fatalities, most often
do not bite. However, human fatalities are on record.
Other snakes found in the sea/ on the sea shore/ in brackish
water and without flat, oar-like tails (Subfamily Homalopsinae)
may bite, but are not currently known to cause human fatalities.
They are thought to be mildly venomous.
Modern Snakebite
First Aid for the Wilderness Environment
Ian D Simpson
W.H.O. Snakebite Treatment Group
Tamil Nadu Government Snakebite Taskforce
Introduction
Today snakebite continues to be a major medical problem in
India. The W.H.O. publish statistics that state that the death
toll could be as high as 50,000, although many Indian
authorities believe the number to be higher.
There are a great many contributory factors as to why the death
toll remains so high. Some argue that one of the major factors
is the reliance by victims on traditional medicines, which have
no proven value in the treatment of snakebite, may be
potentially harmful and are based on ideas superseded by modern
science.
Traditional medicine is believed to be ineffective by the
allopathic tradition because it is perceived to be simply the
accumulation of accepted wisdom, not subject to rigorous
scientific review. However, snakebite has been such a persistent
medical problem in India that there is a danger that similar
accumulated concepts have entered the allopathic tradition and
remain part of the way we regard snakebite in modern India
today. What do we know about snakebites and what is the most
effective first-aid in the event of snakebite?
Venom Action
Snake venom works in essentially two ways, either by
interrupting the nervous system (Neurotoxic) or by attacking the
blood system (anti-haemostatic). In addition, it can cause local
damage as a result of elements in the venom designed to help it
spread throughout the body.
In simple terms Cobras and Kraits are neurotoxic whereas
viperine snakes are anti-haemostatic; however this is
complicated by the fact that the Russell’s Viper can also cause
neurotoxic symptoms. Cobras and Kraits have venom that is
designed to stop nerve impulses from the brain reaching muscles
and hence paralysing them. Once the diaphragm is affected, the
victim dies of suffocation as without the diaphragm the victim
cannot breathe. In these cases keeping the victim breathing
until arrival at hospital will be life saving.
Viper venom is pro-coagulant, it causes the blood to begin
clotting in the human body. Other systems in the body are
designed to combat any unauthorised clotting and they battle the
clotting until eventually one of the factors necessary for the
blood to clot is completely used up. The blood is now
incoagulable and will not clot even if the body activates the
clotting mechanism. In addition, viper venom contains other
enzymes whose function is to damage the blood vessels by
punching holes in them. The victim will now start bleeding and
if not treated will bleed to death.
Signs and Symptoms of Venomous Bite
Viperine Envenoming
Swelling at the bite site (in many but not all cases)
Continual bleeding from the bite site.
Bleeding from the gums or nose
Unusual bruising appearing away from the bite site
Blistering is sometimes seen
Neurotoxic Envenoming
Drooping eyelids
Difficulty speaking, opening the mouth or protruding the tongue
Difficulty supporting the neck and head
Difficulty swallowing
Difficulty breathing
Equipping Yourself for Snakebite in the Wilderness
Environment
Normally you will be entering the wilderness with some basic
first aid equipment. For snakebite, some simple bandages and a
splint for immobilizing the wound will be sufficient. Both these
items can also be improvised.
In addition, for organized groups it would be advisable to take
20 vials of polyvalent anti snake venom (ASV) and an ambubag.
The ASV is to be given to the hospital on arrival, in case they
do not have their own stock, it must not be administered outside
a hospital or by other than a doctor. ASV can cause rapid life
threatening reactions which require other drugs to cure.
In the case of neurotoxic bites the greatest threat is
respiratory failure. The patient will appear dead but as long as
you keep them breathing, will survive until you reach hospital
and the doctor can take over. The ambubag allows the victim to
be kept breathing on the way to hospital.
First Aid Treatment: What You Should
Do!!!!!
Do it R.I.G.H.T.
The preferred method of first aid currently being recommended is
based around the mnemonic “Do it R.I.G.H.T”.
It consists of the following:
R. = Reassure the patient. 70% of snakebites are from non
venomous
species and only 50% of bites by venomous species actually
envenomate the patient.
I. = Immobilise as you would for a fracture without compression.
G. H. = Get to Hospital fast.
T. = Tell the doctor of any symptoms such as drooping eyelids
that
manifest on the way to hospital.
There is no need to wash the wound and this can also lead to
increasing venom flow into the body. Keep the victim as immobile
as possible and transport quickly to hospital.
First Aid Treatment: What You
Should Not Do!
Tourniquets!
In modern India the tradition of using a tourniquet remains and
in addition a new variant has been introduced, that of the
pressure bandage. How useful are these techniques and what is
the science behind them?
Tourniquets are tight constriction devices made of cloth, rope,
string or rubber which are tied around the upper part of the
limb, above the bite, in order to stop the flow of blood into
the limb. Tourniquets tied on the lower part of the limb are
ineffective because the lower limb consists of two bones and it
is therefore difficult to compress the artery sufficiently to
stop or reduce flow. Tourniquets also have the following
drawbacks.
Firstly they are often justified as being necessary when the
victim has considerable distances to travel to the hospital.
However tourniquets left in place for greater than 30 minutes,
cut the oxygenated arterial blood supply and thus place the limb
in danger of ischaemia often resulting in amputation. Secondly,
the vast majority of Indian snakes have venom which causes
significant local tissue damage or necrosis. Using tourniquets
simply confines the toxin to a smaller area and by raising the
intracompartmental pressure within the limb, makes this toxin
more effective and therefore creates greater local tissue damage
and necrosis.
The third area of concern is the consequences of what happens
when the tourniquet is released. In the case of viper bites the
first action of the toxin is pro-coagulant which causes the
blood to begin clotting. The blood below the tourniquet will be
under tremendous pressure to clot and small clots will form in
the bloodstream. Once the tourniquet is released there is a
great danger that these clots will then enter the main part of
the body and cause embolism and death.
The final issue with tourniquets is whether or not they are
effective at all. Research carried out in Burma in the 1980s
showed that venom levels on both sides of the tourniquet were
similar. Further research showed that in 33% of cases where
tourniquets had been applied the victim underwent systemic
envenomation even whilst the tourniquet was still in place,
demonstrating quite clearly that the tourniquet doesn't do what
we believe it does, which is to stop the flow of venom into the
body. It is also interesting to note that in the early 1900’s
when Wall wrote his great work ‘The Poisonous Terrestrial
Snakes’ he dismissed the use of tourniquets on the grounds of
effectiveness. Clearly some myths certainly have the tenacity to
endure despite all evidence to the contrary.
Pressure Immobilisation
Technique!
The modern variation of the tourniquet is the pressure bandage,
Pressure Immobilisation Method or Sutherland Wrap as it has been
called. A great many books, particularly those published by
herpetologists, recommend this method in the event of a bite.
The instructions are to tie a crepe bandage in the same way that
you would for a sprain, tightly enough such that you can place
to two fingers under the bandage.
Again let us look at the background to this technique. This
procedure was derived from work carried out by Sutherland in
1979 in Australia. Sutherland's work appeared to demonstrate
that the rate of flow of venom into the body could be reduced by
the application of this technique. However Sutherland’s research
work was carried out on monkeys tied to wooden boards and thus
did not represent a practical setting. Sutherland also
recommended that for the technique to be successful an integral
splint had to be included with the bandage. Sutherland himself
did not recommend the version currently carried out in India of
simply applying the bandage without a splint.
Finally, Sutherland concluded, to be effective the level of
pressure that the bandage had to apply was the equivalent of 55
mm of mercury. Further research carried out in Australia
demonstrated that to be effective, the range of pressure in the
upper limb had to be between 40 and 70 mm of mercury and in the
lower limb 55 to 70 mm of mercury.
In addition, this research demonstrated that even if the
technique had been accurately applied, the simple act of walking
for 10 minutes rendered the technique ineffective. Also pressure
bandages above the recommended level of pressure may in fact
increase the flow rate of venom into the system.
Later work carried out in the United States demonstrated that
doctors carrying out the technique were only able to achieve the
correct level of pressure 13% of the time and lay people 5% per
cent of the time.
The lack of practicality in the application of this technique in
India is compelling.
Other Methods
Other methods have been proposed such as cutting the wound to
release the venom. Unfortunately evidence has shown you cannot
bleed venom from a wound. In cases of viper bite, when the blood
becomes incoagulable, cutting the victim will cause
uncontrollable bleeding and is life threatening.
Evidence has shown you cannot suck venom from a wound, wither by
mouth or by using a mechanical suction device. Mechanical
suction devices, advertised in the U.S. do not remove venom and
may make matters worse by causing greater local tissue damage.
The use of electrical shocks to denature the venom was also
popular at one time in the U.S. Research has shown that electric
shocks DO NOT denature venom and simply electrocute the victim!
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